丘脑血供

丘脑的血液供应非常有意思，看看这一个病例几乎能够全面了解丘脑的血供。 Figure 1. T2 weighted magnetic resonance imaging with axial planes (a–c) shows infarcts affecting all four arterial territories of the thalamus (for details see text) and temporo-occipital areas supplied by the right posterior cerebral artery, a lacuna adjacent to the lateral part of the anterior limb of the right internal capsule, and dilated Virchow–Robin spaces in both basal ganglia. Figure 2. Three-dimensional time-of-flight magnetic resonance angiography with coronal (a) and axial (b) planes shows no posterior communicating artery and an occlusion of the right posterior cerebral artery (arrowheads), that begins 7 mm distal to its origin from the basilar artery. 丘脑与行为： Figure 1. Schematic view of the arterial supply to the thalamus. (A) Lateral view; (B) view from above; (C, D) detailed relationship between the arterial territories and nuclear subgroups within the thalamus. 1 =carotid artery; 2 =posterior communicating artery; 3 =basilar artery; 4 =thalamogeniculate arteries; 5 =tuberothalamic artery; 6 =posterior choroidal artery; 7 =paramedian pedicle; 8 =posterior cerebral artery. Main thalamic nuclei and tracts: CL =central lateral; CM =centromedian;Co =commissural; Cp =commissural posterior;DM =dorsomedian; MTT =mamillothalamic tract; Pua=pulvinar anterior; Pum =pulvinar medial; Pul =pulvinar lateral; Pf =parafascicularis; R =reticular; VA =ventral anterior; VLa = ventral lateral anterior; VLp =ventral lateral posterior; VPLa = ventroposterolateral anterior;VPLp = ventroposterolateral posterior; VPM =ventroposteromedian. Figure 2. (A) Diffusion-weighted MRI of a left anterior thalamic infarct involving the anterior nuclei and mamillothalamic tract in a 58-year-old patient who presented with perseverations, incoherent speech with intrusion of previous topics, and distorted memories. (B) Schematic representation (see figure 1 for legend) Figure 3. (A) T2-weighted MRI showing a bilateral paramedian stroke predominantly on the left side in a 39-yearold woman. This patient presented with improper behavior and inadequate and disinhibited comments to her husband.Three hours later, she complained she was “not able to control her eyes,” then rapidly became comatose. Cognitive tests, performed 3 days later, showed persistent loss of self-activation and severe amnesia. (B) Schematic representation (see figure 1 for legend). Figure 4. (A) T2-weighted MRI of a unilateral left paramedian infarct in a 54-year-old man who, during coronary angiography, presented sudden vertical diplopia and transient hypersomnolence. Cognitive tests, performed 2 days later, were entirely normal except for loss of selfactivation.(B) Schematic representation (see figure 1 for legend). Figure 5. (A) T2-weighted and (B) diffusion-weighted MRI,showing bilateral venous infarct predominantly on the right side in a 18-year-old woman who, 3 days after unusualhead aches, presented a decreased level of consciousness. Cognitive tests showed a lack of self-activation, severe attentional deficits, anosognosia, severe verbal and visuospatial amnesia,and a diminished capacity for abstraction and problem solving. (C) Venous angio-MRI showed absence of the sinus rectus and deep cerebral veins. Figure 6. (A) T2-weighted MRI of a left inferolateral thalamic infarct in the territory of the thalamogeniculate arteries in a 58-year-old man showing executive dysfunction with verbal fluency difficulties, pathologic response inhibition,and pathologic conceptual ability, in addition to a right sensory and ataxic hemisyndrome. (B) Schematic representation (see figure 1 for legend).